Further studies focused on the subpopulations of HSPCs isolated from either BM samples from patients with MM or G-CSF-mobilized healthy donor leukopaks, including long-term HSPCs, short-term HSPCs and multi-potent progenitor cells, showing that all three HSPC subpopulations expressed undetectable CD1d and were not subjected to Allo15BCAR-NKT cell killing (Supplementary Fig. 12a–j). This evidence concerns the gene CSF3 and Miyoshi myopathy.