In an animal model of experimental autoimmune encephalomyelitis (EAE), which is the most commonly used rodent’s model of MS, chronic T. gondii infection modulates the immunological response towards the production of anti-inflammatory cytokines (i.e. interleukin-10; 34) and prevents blood–brain barrier (BBB) disruption caused by EAE 35 what seem to support the hypothesis about the protective effect of Toxoplasma infection on the development of autoimmune disorders. Here, IL10 is linked to experimental autoimmune encephalomyelitis.