Subsequent NGS testing of the primary tumor revealed the presence of EML4 (exon7)-ALK (exon20) fusion, which is notably distinct from EML4-ALK V1 (exon 13 of EML4 fused to exon 20 of ALK) or V3 (exon 20 of EML4 fused to exon 20 of ALK) variants. Here, EML4 is linked to neoplasm.