Recent studies have shown that IFN-α and IFN-γ are involved in the aggregation of infiltrating immune cells in the salivary glands of SS patients through the Janus kinase (JAK)-signal transducer and activator of transcription (STAT) signaling pathway by promoting the production of CXCL13 and CXCL10, respectively (107, 108). The gene discussed is IFNA1; the disease is synovial sarcoma.