SLITRK4 can rescue neurite outgrowth deficits in myotonic dystrophy type 1 (DM1) patient-derived cells, and its expression is downregulated in the neural cells and brains of DM1 patients (Marteyn et al., 2011), suggesting its clinical importance in DM1. The gene discussed is SLITRK4; the disease is myotonic dystrophy type 1.