We found that CCL7 is increased in IPF and negatively correlated with lung function, consistent with a previous report showing an increase in CCL7 in fibroblasts from patients with usual interstitial pneumonia [49], in serum of patients with systemic sclerosis [50], and a recent preprint showing a significant increase of CCL7 in IPF plasma compared to that of matched controls [48]. This evidence concerns the gene CCL7 and idiopathic pulmonary fibrosis.