On the other hand, 1 μM of CEP tended to inhibit the secretion of IL-6, IL-10, IL-17, TNF-α, and IFN-γ from activated PBMCs (Fig. 7C-G), which would help to relieve theTh1/Th2/Th17 immune responses, since these cytokines were reported to contribute to the pathogenesis of ITP [21]. The gene discussed is IFNG; the disease is autoimmune thrombocytopenic purpura.