To determine the contribution of NEAT1 to aerobic glycolysis in cervical cancer cells, we detected the levels of glucose, lactate and ATP in cervical cancer cells and found that NEAT1 knockdown led to decreased glucose consumption, lactate production as well as ATP production in HeLa cells, whereas NEAT1 overexpression led to the opposite results (Figure 2A–C). The gene discussed is NEAT1; the disease is cervical cancer.