Regarding the involvement of CCL9 in pancreatic cancer and pancreatitis, in addition to our results shown here and as mentioned previously [46], another recent study has reported that pancreatic cancer cells of Pdx-1cre:KrasG12D:TP53R172H (KPC) mice upregulated CCL9 to recruit granulocytic myeloid-derived suppressor (G-MDSC) cells as an alternative mechanism to resist to drug-induced cell death, even when a higher CD8+ T cell response was induced by drug treatment [57]. This evidence concerns the gene CD8A and pancreatitis.