Similarly, previous studies conducted by our group demonstrated that EBV DNA increases the production of IL-17A, a proinflammatory cytokine that has been extensively studied for its association with inflammation and autoimmune diseases, while the inhibition of Toll-like receptors (TLRs) 3, 7, and 9 significantly reduces the EBV DNA-mediated triggering of IL-17A production in mice [16,17]. The gene discussed is IL17A; the disease is autoimmune disease.