Prostate cancer (PCa) cells can uptake miR-95 delivered by TAM-derived exosomes, and the binding of miR-95 with its downstream target gene JunB promotes PCa cell invasion, proliferation, and epithelial-mesenchymal transition (EMT); moreover, the high miR-95 expression level is associated with worse clinicopathological features [92]. The gene discussed is JUNB; the disease is posterior cortical atrophy.