In contrast, we found that these signaling interactions are significantly altered in RasV12,scrib− cells, as Wg, Dronc, JNK, and Yki are all robustly upregulated (Figure 1 and Figure S1), suggesting a non-apoptotic tumor growth-promoting role for JNK and Dronc and growth-promoting mitogenic roles for Yki and Wg. Here, YAP1 is linked to neoplasm.