TENT5C and laryngotracheoesophageal cleft: Apart from the previously described study by Xia and colleagues, in which the authors suggested FAM46C to be the downstream target of three different lung SCC-specific onco-miRNAs and hence responsible for inhibiting proliferation of cells derived from SCC of the lung [61], Li et al. hypothesized FAM46C to be a major player in LC progression.