NLRP3 and myocardial infarction: In summary, this study supported that IT obstructed the TGF-β1 pathway activation to reduce oxidative stress in myocardial tissues of MI rats, and down-regulate the activation of the NLRP3 inflammasomes, thus improving the cardiac function of MI rats, which provided some theoretical basis for clarifying the pathogenesis of MI and novel ideas for MI therapy.