CXCL9 and cervical cancer: Furthermore, genetic knockout of K17 in a mouse papillomavirus (MmuPV1) model of cervical cancer results in rapid regression of papillomas and increased CD8+ T cell infiltration [59] whereas K17 expression promoted the expression of pro-inflammatory cytokines IFN ‐γ, CXCL9, CXC110, CXC111, TNF-α, and TGF-β, among others [65].