In basal cell carcinoma of the skin (BCC), increased K17 expression correlates with the expression of key pro-inflammatory chemokines such as CXCR3 andCXCL10, among others [5] and the genetic ablation of K17 delays BCC onset in mouse models that correlates with a global cytokine switch that differentially regulates T cell infiltration [14]. The gene discussed is CXCR3; the disease is skin basal cell carcinoma.