GPX4 and gastric cancer: To explore whether baicalin could regulate the occurrence of ferroptosis in gastric cancer through p53 or not, baicalin of 200 μM and 250 μM combined with PFN-α of 10 μM were used to treat HGC27/L cells for 24 h, and the inhibition rate of GSH, the expressions of genes p53 (Fig. 5g), SLC7A11, GPX4 (Fig. 5h), and ROS changes (Fig. 5l, m) were is needed.