NMNAT3 and rheumatoid arthritis: Inhibition of NMNAT3 by tannic acid led to a reduction in cell proliferation, suggesting that tannic acid might be a potential effective treatment for RA patients (Table 2) (65).Togher with data from T1D/SLE-iPSCs-derived cardiomyocytes, we may conclude that diseased cardiomyocytes show more or less abnormal phenotypes although these cells can express normal cardiomyocyte marker, suggesting cardiac complications are a key preventive disease for patients of autoimmune diseases.