Moreover, in the same dataset, INPP4A expression levels showed moderate to strong correlation with expression of various well established pro‐fibrotic marker genes such as MUC5B, ACTA2, COL1A1, EPCAM, and FN1 (Figure S3) that were differentially expressed between IPF fibroblastic foci and control alveolar septae (Figure S4). The gene discussed is EPCAM; the disease is idiopathic pulmonary fibrosis.