Moreover, in the same dataset, INPP4A expression levels showed moderate to strong correlation with expression of various well established pro‐fibrotic marker genes such as MUC5B, ACTA2, COL1A1, EPCAM, and FN1 (Figure S3) that were differentially expressed between IPF fibroblastic foci and control alveolar septae (Figure S4). This evidence concerns the gene COL1A1 and idiopathic pulmonary fibrosis.