Together, these results suggest that INPP4A expression follows a non‐linear pattern through the disease course, and the increased expression observed in bulk IPF lungs might be a reflection of INPP4A expression in lesser fibrotic regions or seemingly unaffected areas of IPF tissues than the severe fibrotic regions such as fibroblastic foci. Here, INPP4A is linked to idiopathic pulmonary fibrosis.