Research indicates that GBAP1, stimulated by METTL3, facilitates the movement, penetration, and multiplication of HCC cells through the GBAP1/miR‐22‐3p/BMPR1A/SMAD pathway, hinting at the role of GBAP1 as a possible prognostic marker and target for HCC treatment.10 Here, BMPR1A is linked to hepatocellular carcinoma.