Cytokines previously described in the literature as associated with senescence (TNFα, IL-6, VEGF, IL-1RA, GM-CSF, CCL20, CXCL8, IL-1β, CXCL1, CXCL2, G-CSF, CCL2 and IL-10) [16, 17, 29, 30] or with the recruitment of myeloid cells (CXCL10, fractalkine and IL-33) were compared between COVID-19+ and control cohorts. The gene discussed is CCL20; the disease is COVID-19.