Deletion of RELMα from either CC10+ or CD11c+ cells alone was insufficient to result in statistically significant changes; however, constitutive RELMα KO mice had increased levels of alveolar destruction at day 30 post infection, in line with previous studies conducted at earlier time points (Chen et al., 2016; Batugedara et al., 2018; Sutherland Id et al., 2018), which suggests a tissue-protective role for RELMα during helminth infection that can be derived from either epithelial cells or CD11c+ cells. Here, ITGAX is linked to infection.