A recent study showed that 16 cases of enteric and mucinous adenocarcinoma lacked common driver mutations except for KRAS mutations [43], indicating that mucinous, enteric, and colloid adenocarcinomas might form a single spectrum of HNF4α-positive non-TRU-type adenocarcinomas showing gastrointestinal differentiation. The gene discussed is HNF4A; the disease is adenocarcinoma.