Study has demonstrated that variations in the CD36 gene can exert a significant influence on an individual’s susceptibility to cardiovascular illnesses,[16] type 2 diabetes,[17] and metabolic disorders.[18] Additionally, research has revealed that the signaling of oxidized LDL/CD36 in macrophages establishes a connection between dysregulated fatty acid metabolism and oxidative stress from the mitochondria. The gene discussed is CD36; the disease is Other metabolic disease.