Immunological (CD4+ cells, CD8+ cells, and CD4+/CD8+), virological (plasmatic HIV viremia), serological parameters for several infections (presence of CMV IgG, HCV antibodies, HBsAg, HBcAb or Toxoplasma IgG), and the number of comorbidities did not appear to alter the risk of PCCs in our cohort. The gene discussed is CD4; the disease is infection.