Another research has shown a novel mechanism by which mucin-type core 3 O-glycan influences the epithelial-mesenchymal transition (EMT) and mesenchymal-epithelial transition (MET) plasticity of colorectal cancer (CRC) cells through a MUC1/p53/miR-200c-dependent signaling cascade (66). This evidence concerns the gene MUC1 and colorectal carcinoma.