ILK and myocardial infarction: Different isoforms of integrins and their associated proteins and appropriate mechanical signaling can promote cardiac repair after myocardial injury, especially myocardial infarction, involving gene therapy (ILK, α7β1D), modified cardiac progenitor/stem cells, endothelial progenitor cell transplantation, bone marrow mesenchymal stem cells (BMSCs), outer matrix transplantation therapy (β1, ILK), cell-free therapy with stem cell-derived paracrine factors (thymosin β4, periostin), non-invasive assessment of angiogenic αvβ3 molecular imaging, and cardiac tissue engineering.