A study by Mao et al. showed that miR-200c-3p was highly expressed in an atherosclerosis (AS) mouse model established by feeding ApoE−\/- mice with a high-fat diet, and promoted the process of ox-LDL induced EMT in human umbilical vein endothelial cells (HUVECs) by suppressing the SMAD7/YAP pathway, thereby promoting the transformation of ox-LDL-treated HUVECs from a cobblestone-like epithelial phenotype to a spindle-like mesenchymal phenotype, which may be involved in atherosclerosis (Mao and Jiang, 2021). The gene discussed is YAP1; the disease is atherosclerosis.