Lai et al. [33] report that GTSE1 positively regulated the transcriptional level of FOXM1, downstream factors of FOXM1 (CCNB1 [34] and CCND1 [35]), and transcription factors of FOXM1 (HIF-1α [36], SP1 [37], and E2F1 [38]), and shFOXM1 or the FOXM1 inhibitor reverses the pro-proliferation effect of GTSE1 in prostate cancer. Here, E2F1 is linked to prostate cancer.