Further studies found that CCT significantly reduced the activities of ALT and AST, as well as the levels of TG, creatinine, malondialdehyde, TC, blood urea nitrogen, and uric acid, which prevented further exacerbation of NAFLD, and its main mechanism of action was closely related to the upregulation of HO-1 and Nrf2 expression, the reduction of inflammation, and the inhibition of oxidative stress. Here, GPT is linked to metabolic dysfunction-associated steatotic liver disease.