Although the genes analyzed in our study have been associated with monogenic and multifactorial traits (coronary artery disease, immunodeficiency 100 with pulmonary alveolar proteinosis and hypogammaglobulinemia, resistance to HIV-1, susceptibility to Mycobacterium tuberculosis and spina bifida, among others), no correlation was found between CCL2 and any COVID-19-related comorbidities in a GWAS that included inflammatory protein-coding genes in older males (Hillary et al., 2020). The gene discussed is CCL2; the disease is COVID-19.