Subsequently, the authors constructed an in vivo infection model and reported that the administration of TLR2 inhibitors after infection modestly improved survival in mice and significantly reduced the release of proinflammatory cytokines such as IL‐6, tumor necrosis factor(TNF), and IFNs, indicating that TLR2‐mediated inflammation is pathogenic in severe acute respiratory syndrome coronavirus 2(SARS‐CoV‐2) infection.97, 98, 99. The gene discussed is TNF; the disease is infection.