RSV infection also induces STAT1-dependent IFNγ production by NK cells, while STAT1-deficient mice exhibit increased IL-5+ and IL-13 + ILC2s and IL-17A + ILC3s in the lung in response to RSV infection [170], suggesting that ILCs play a role in RSV infection beyond impacting asthma pathology. The gene discussed is IL5; the disease is asthma.