The latter have been found to partially induce leukemic cell apoptosis through activation of intrinsic and extrinsic apoptosis pathways in an acute promyelocytic leukemia cell line NB4 by significantly increasing the expression of pro‐apoptotic genes BID and BAX, while decreasing the expression of the anti‐apoptotic gene BCL2 [56]. The gene discussed is BCL2; the disease is acute promyelocytic leukemia.