TGFB1 and Fabry disease: Given the relatively small size of leukocyte infiltrates in most FD (compared to IMID), and the predominance of myeloid cells and granulocytes, an obvious question is: does combining TGFβ with pre-existing alarmin, PRR or serum signaling suppresses the production of M-CSF, GM-CSF or G-CSF by fibroblasts?