Compared with the control group, they found that CDT treatment reduced the cones in the hippocampus and CA3 area, Cell damage, IL‐6 and TNF‐α expression and average escape latency, increase ChAT (Ach biosynthetic enzyme), activated C kinase 1 (RACK1)/BDNF, increase the time spent in the target quadrant and the number of platform crossings. Therefore, CDT could balance cytokines and nutritional factors and improve cognitive defects caused by neuroinflammation.120. The gene discussed is BDNF; the disease is Cognitive impairment.