MAPT and Alzheimer disease: Currently, attempts are being made to develop organoid models of the disease using multi‐tissue organoid models including astrocytes, oligodendrocytes, microglia, and neurons;72 organoid models developed using multiple tissues more closely resemble the pathological processes of AD, including tau protein hyperphosphorylation, neuroinflammatory activity, harmful Nitrogen Monoxide (NO) release to AD astrocytes and neurons, axonal segment fracture caused by neurotoxic activity, microglia recruitment, and β‐amyloid aggregation.73, 74