In the seventh most cited article, i.e., the study by Zhou et al., it was indicated that circRNA_010,567 silences upregulates miR-141, downregulates TGF-β1 expression, and inhibits fibrosis-associated proteolytic excision in CFs (including Col I, Col III, and α-SMA), which represents a novel role of circRNAs in the pathogenesis of central myofibrillar fibrosis [70]. Here, ACTA1 is linked to fibrosis.