On the other hand, GRPR is proven to have higher expressioninthe early stages of PCa, which is even retained on metastatic sites.15−17 The inverse correlation between the two biomolecules indicates thatnot only both are valid as targets for PCa therapy and/or diagnosis(theranostics) but also they are complementary to each other.18,19. This evidence concerns the gene GRPR and posterior cortical atrophy.