Interestingly, WIN 55, 212-2 (a synthetic ligand of CB1) treatment in glioma cells, and URB597, a FAAH1 inhibitor, also arrested at G0/G1 only [35], suggesting a link between CB1 stimulation–FAAH1 inhibition and G0/G1 arrest, which is to be investigated. Here, CNR1 is linked to central nervous system cancer.