However, AV presents a unique profile, marked by a more pronounced increase in interleukin 1β (IL-1β) compared to BV, combined with elevated levels of interleukin 6 (IL-6) and interleukin 8 (IL-8), which are not commonly seen in BV cases [35,36,37]. The gene discussed is CXCL8; the disease is bacterial vaginosis.