Furthermore, the treatment with MT is capable of modulating glucose homeostasis because it functions as a glycolytic agent since it inhibits the Warburg effect that is present after the hijacking of mitochondrial function by the SARS-CoV-2 virus and is characterized by elevation of lactate dehydrogenase, lactic acidosis, and Cyt c and COX II liberation that promotes hyperferritinemia. This evidence concerns the gene MTCO2P12 and lactic acidosis.