By researching lncRNA Nuclear Paraspeckle Assembly Transcript 1 (NEAT1) it was found that it was responsible for the promotion of allergic rhinitis (AR) pathogenesis via EVs, which interacted with human nasal epithelial cells (HNECs) and induced IL-13 production and apoptosis, which can lead to damage to the epithelium and increase the likelihood of allergen exposure and development of allergic sensitization [22]. The gene discussed is IL13; the disease is allergic rhinitis.