Knockdown of miR-218 in MSCs partially abrogated the beneficial effects of MSC-Exos, while transfection of miR-218 and its overexpression in MSC-Exos resulted in increased BMP2 expression, which led to the attenuation of EndMT and alleviation of pulmonary fibrosis [55]. This evidence concerns the gene BMP2 and pulmonary fibrosis.