Xie and colleagues revealed that BM-MSC-Exo-sourced miR-214 targeted IL-33 and blocked the IL-33/ST2 axis in fibroblasts and lung-infiltrated immune cells, which reduced collagen fiber accumulation and α-SMA production, resulting in the attenuation of bleomycin-induced pulmonary fibrosis in experimental mice [56]. This evidence concerns the gene IL33 and pulmonary fibrosis.