Comparing shared genetics in uterine endometria, endometriosis, and EAOC, almost half of clear-cell carcinomas and endometrioid carcinomas harbored mutations in the tumor-suppressor gene ARID1A (AT-rich interactive domain-containing protein 1A) [46], encoding BAF250a—a main component of the ATP-dependent chromatin remodeling complex SWI/SNF involved in the transcriptional activation of chromatin-repressed genes [49]. This evidence concerns the gene ARID1A and clear cell adenocarcinoma.