MDS-like AML blasts have higher expression of HLA-II, LGALS9, and TGFB1, while T- and NK cells display elevated levels of their cognate receptors LAG3, HAVCR2, and TGFBR3 and secrete more IFNγ, compared to non-MDS-like AML [79]. This evidence concerns the gene TGFB1 and acute myeloid leukemia.