For example, dyslipidemia is strongly linked with H. pylori/MetS, and MC activation, through the release of tryptase and chymase, contributes to dyslipidemia [141], while eradication of H. pylori improves lipid profiles such as fibrinogen, an independent risk factor for MetS-related C-CVD and dementia [25,142]. This evidence concerns the gene CMA1 and metabolic syndrome.