Namely, Apert syndrome is caused by a mutation in FGFR2 and includes phenotypic characteristics like cleft palate, hearing loss, and hypertelorism, while Crouzon syndrome is a result of FGFR2 and FGFR3 mutation, presenting with mandibular prognathism, tarsal bones’ fusion, and hearing loss. The gene discussed is FGFR2; the disease is Crouzon syndrome.