Glomerular lipidosis is presented not only by diseases involving genetic abnormalities in lipid metabolic pathways, such as lipoprotein glomerulopathy (LPG) or lecithin-cholesterol acyltransferase deficiency, but also those accompanied by intraglomerular infiltration of lipid-laden macrophages/histiocytes. This evidence concerns the gene LCAT and lipoprotein glomerulopathy.