Several studies have linked PE and HELLP to dysregulated hemostasis and thrombotic microangiopathies (TMAs), which are characterized by elevated levels of the von Willebrand factor antigen (vWFAg) and reduced plasma activity of ADAMTS13 (a disintegrin and metalloproteinase with a thrombospondin type 1 motif, member 13) [2,6]. This evidence concerns the gene ADAMTS13 and Genetic thrombotic microangiopathy.