To validate the docking prediction and assess whether EDA-mediated AHR pathway induction could be conserved in a human experimental model, we assessed the ability of EDA to induce the nuclear translocation of AHR and subsequent expression of endogenous AHR target genes in the neuroblastoma cell line SH-SY5Y, which represents a relevant cellular model for investigating this signaling pathway [45]. This evidence concerns the gene AHR and neuroblastoma.